TY - JOUR
T1 - Cardiac amyloidosis, constrictive pericarditis and restrictive cardiomyopathy
AU - Meaney, Eduardo
AU - Shabetai, Ralph
AU - Bhargava, Valmik
AU - Shearer, Mary
AU - Weidner, Cathy
AU - Mangiardi, Lucia Maria
AU - Smalling, Richard
AU - Peterson, Kirk
N1 - Funding Information:
From the Cardiology Section, Veterans Administration Hospital, and the Department of Medicine, University of Kentucky College of Medicine, Lexington, Ky. 40506. This study was supported in part by funds from Veterans Administration Research Grant and Tobacco and Health Grant 124-05-7H350-21066. Manuscript received January 2, 1976; revised manuscript received April 12, 1976, accepted April 14, 1976. t Hospital del Norte, Mexico City, Mexico. ~; University of California at San Diego, San Diego, Calif. * Present address and address for reprints: Ralph Shabetai, MD, Cardiology Section (IliA), Veterans Administration Hospital, 3350 La Jolla Village Dr., San Diego, Calif. 92161.
PY - 1976/11/4
Y1 - 1976/11/4
N2 - Cardiac amyloidosis is not characterized by a single hemodynamic pattern. Some of the cases present the clinical findings of restrictive cardiomyopathy and in these differentiation from constrictive pericarditis remains difficult in spite of the introduction of techniques designed to assess myocardial contractility and ventricular diastolic compliance. The clinical features and the demonstration of left ventricular diastolic pressure greater than right remain the most useful means of distinguishing restrictive cardiomyopathy from constrictive pericarditis. In other cases of cardiac amyloidosis the diastolic pressure is elevated throughout diastole and ventricular ejectile ability is lost. These cases do not simulate constrictive pericarditis and should not be classified as restrictive cardiomyopathy.
AB - Cardiac amyloidosis is not characterized by a single hemodynamic pattern. Some of the cases present the clinical findings of restrictive cardiomyopathy and in these differentiation from constrictive pericarditis remains difficult in spite of the introduction of techniques designed to assess myocardial contractility and ventricular diastolic compliance. The clinical features and the demonstration of left ventricular diastolic pressure greater than right remain the most useful means of distinguishing restrictive cardiomyopathy from constrictive pericarditis. In other cases of cardiac amyloidosis the diastolic pressure is elevated throughout diastole and ventricular ejectile ability is lost. These cases do not simulate constrictive pericarditis and should not be classified as restrictive cardiomyopathy.
UR - http://www.scopus.com/inward/record.url?scp=0017103745&partnerID=8YFLogxK
U2 - 10.1016/S0002-9149(76)80001-X
DO - 10.1016/S0002-9149(76)80001-X
M3 - Artículo
SN - 0002-9149
VL - 38
SP - 547
EP - 556
JO - The American Journal of Cardiology
JF - The American Journal of Cardiology
IS - 5
ER -